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Associate Professor of Molecular Physiology and BiophysicsAssociate Professor of
Primary Office: 6-510 BSBIowa City, IA 52242
Primary Office Phone: 319-335-7886
Email: firstname.lastname@example.orgWeb: Henry Laboratory
BS, Genetics, University of GeorgiaPhD, Biology, Massachusetts Institute of Technology
Biosciences Graduate ProgramDepartment of Molecular Physiology and Biophysics PhDInterdisciplinary Graduate Program in Translational BiomedicineMedical Scientist Training Program
The long term research goals of my laboratory are to understand the molecular and cellular basis of prostate cancer progression and metastasis in order to develop new methods for the diagnosis and treatment of this disease. Current efforts are focused on the role of a cell-matrix receptor dystroglycan and epithelial-mesenchymal transition in this process. We approach this problem using both cell-based and animal models of disease progression. Emerging interests include how physiological and environmental components interact with central genetic pathways related to disease progression.
Cardiovascular Research CenterCenter for Gene Therapy of Cystic Fibrosis and other Genetic DiseasesHolden Comprehensive Cancer Center
Cellular and Molecular Biology of Cancer Cell Extravasation.
2012. Norian L,
Eradication of Metastatis Renal Cell Carcinoma after Adenovirus-Encoded TNF-Related Apoptosis-Inducing Ligand (TRAIL)/CpG Immunotherapy.
Glycosylation of alpha-dystroglycan in prostate cancer: Prognostic implications and regulation by LARGE2.
2012. Nauseef J,
Resistance to fluid shear stress is a conserved novel biophysical property of malignant cells.
2012. Tang X,
A critical role of GBy in tumorigenesis and metastasis of breast cancer.
J. Biol. Chem..
Characterization of an inducible mouse model of abacterial prostatitis.
Down Regulation of CSL Inhibits Cell Proliferation in Prostate and Breast Cancer Cells.
J. Cell. Biochem..
Epithelial-to-mesenchymal transition in prostate cancer: Paradigm or puzzle?.
Nature Reviews Urology.
Slow disease progression in a C57BL/6 Pten deficient mouse model of prostate cancer.
Am. J. Pathol..
Resistance to fluid shear stress is a conserved biophysical property of malignant cells.
Date Last Modified: 06/04/2015 -
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